Many children should NOT receive vaccinations because they have underlying mitochondrial disorders that make them exponentially more vulnerable to vaccine damage. Mitochondrial disorder results from alterations in DNA that damage the body’s energy system, including the ability to produce and utilize ATP (Adenosine Tri-Phosphate). This changes the way the immune system responds to challenge and results in systemic damage rather than the child responding with increased immunity (which is the supposed goal of vaccinations).
Here is a link where you can get information about mitochondrial disorder:
Mitochondrial disorder may be passed from mother to child. It can also happen at any time during the lifespan as a result of exposure to toxins. Mitochondrial disorder causes a multitude of illnesses, including diabetes, thyroid disorders, chronic fatigue syndrome, fibromyalgia, and autism. ANYONE who has these issues needs to be tested for mitochondrial disorder before being vaccinated. Any mother who has these issues needs to know that her child is at increased risk of vaccine injury if he or she has an underlying mitochondrial disorder that has not been diagnosed.
In addition to family history, there are other things that increase the likelihood of a child developing mitochondrial disorder.
Infants and children living in areas where there are high levels of environmental toxins are at increased risk of mitochondrial disorder. If you live near coal-burning power plants or in areas where there are lots of manufacturing businesses or farms that utilize pesticides and/or large amounts of fertilizer, your child should be tested for mitochondrial disorder before vaccination. Some may say this is an overly zealous recommendation. You should know that having mitochondrial disorder increases the risk of vaccine injury to 1 in 50.
If an infant or child was fed soy formula there is increased risk of mitochondrial disorder because of high levels of manganese in the formula. Infants who have been fed soy-based formula should be checked for mitochondrial disorder before vaccination.
Here are some links for information about soy formula, manganese and mitochondrial disorder:
http://sciencelab.com/xMSDS-Manganese-9924577 (see section 11)
http://scholar.google.com/scholar?q=manganese+and+mitochondrial&hl=en&as_sdt=0&as_vis=1&oi=scholart (scholarly articles on manganese and mitochondrial disorder)
http://www.ehjournal.net/content/9/1/48 (VERY recent article about airborne particulate matter, manganese, and mitochondrial disorder)
Just because your child does not have mitochondrial disorder before being vaccinated does not mean he or she won’t have it after vaccination. That’s because the aluminum used as an adjuvent in vaccines causes mitochondrial disorder. So, if your child is vaccinated on the first day of his or her life with the hepatitis B vaccine, which contains high levels of aluminum, and then you have your child tested for mitochondrial disorder, and the test is positive, there’s no way to know where it came from. Unless you are also tested and you test negative.
Here is some information from a recent article (peer-reviewed medical literature) about aluminum and mitochondrial disorder:
In the present review we describe how the use of a systems biology approach in cultured hepatoblastoma cells (HepG2) allowed the identification of the molecular targets of Al toxicity. Mitochondrial metabolism is the main site of the toxicological action of Al. Fe-dependent and redox sensitive enzymes in the tricarboxylic acid (TCA) cycle and oxidative phosphorylation (OXPHOS) are dramatically decreased by Al exposure. In an effort to compensate for diminished mitochondrial function, Al-treated cells stabilize hypoxia inducible factor -1a (HIF-1a) to increase ATP production by glocolysis. Additinoally, Al toxicity leads to an increase in intracellular lipid accumulation due to enhanced lipogenesisi and a decrease in the B-oxidation of fatty acids. Central to these effects is the alteration of a-ketoglutarate (KG) homeostasis. In Al-exposed cells, KG is preferentially used to quench ROS leading to succinate accumulation and HIF-1a stabilization. Moreover, the channeling of KG to combat oxidative stress leads to a reduction of L-carnitine biosynthesis and a concomitant decrease in fatty acid oxidation. The fluidity and interaction of these mtabolic modules and the implications of these findings in liver-related disorders are discussed herein. (citation) (Note: The importance of these findings is relevant especially for brain development and for chronic autoimmune disorders such as diabetes and autism. The disruption of mitochondrial metabolism is significant in the increased risk of vaccine-injury in persons who have underlying mitochondrial disorders – See the Hannah Poling case for more information.)
I hope this is helpful information to anyone who is concerned about whether or not your child is at increased risk of vaccine injury. Please share with anyone you believe will listen (and with those you think won’t – at least you are planting seeds and can sleep with a clear conscience!)
For more information on ingredients contained in vaccines, please read: